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Antibodies reduced disease features in mice with C9orf72-related ALS and FTD

Original source

Antibody Therapy Targeting RAN Proteins Rescues C9 ALS/FTD Phenotypes in C9orf72 Mouse Model.Neuron · 9 December 2019 (opens in a new tab)

Compass summarised this from the study's abstract.

Study details

Studied in
Human, Mouse

Related topics

Researchers developed antibodies targeting RAN proteins produced by the C9orf72 genetic expansion. In cells and C9orf72 mouse models, the antibodies reduced these proteins and signs of toxicity, improved behaviour, reduced neuroinflammation and neurodegeneration, and extended survival.

Why this matters

The findings identify a possible treatment strategy for C9orf72-related ALS and frontotemporal dementia. The work was conducted in cells and genetically modified mice, so it does not yet show a benefit for people or change current treatment.

Limitations and context

This was preclinical research in cells and C9orf72 BAC transgenic mice, not a human clinical trial. The results still need to be tested for safety and effectiveness in people with C9orf72-related disease.

Summarised by Compass 10 August 2026

This summary was generated by AI from the source listed above. It is not medical advice, so read the original source for anything that affects your care.

Bibliographic data from PubMed is courtesy of the U.S. National Library of Medicine. Compass does not reproduce source abstracts and may not reflect the most current record.

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